Luteal Phase Defect: Natural Treatment Options

Luteal Phase Defect: Natural Treatment Options

The third phase of a woman’s menstrual cycle is called the Luteal Phase, and is the time between ovulation and start of a woman’s period. It is during this important time that fertilization and implantation occurs.

As you might imagine, any disruption in the luteal phase of the cycle can be cause for concern since it can affect fertility. Most experts agree that a luteal phase less than 12 days does not give the uterus sufficient time to establish a nourishing lining for a growing fetus and therefore will cause a miscarriage if fertilization has occurred.

Symptoms of Luteal Phase Defect

  • A short menstrual cycle
  • Low progesterone
  • Disrupted body basal temperatures after ovulation
  • Odd symptoms during luteal phase such as low back pain, bleeding and loose stools

Fertility charting can help to determine the length of your luteal phase and if you have low progesterone. Luteal phase defect is estimated to affect 3-4% of women who have ‘unexplained infertility’, up to 63% of women who repeatedly miscarry and 6-10% of women who are fertile.

There are several factors which can cause a luteal phase defect (LPD). The most common is a low progesterone level. Progesterone is an important hormone needed for preserving the uterine lining and pregnancy during the first trimester. In the event a woman’s progesterone production did not reach the optimum level during this important stage in her cycle, a LPD may occur.

Low Progesterone and Luteal Phase Defect

Progesterone is produced throughout the entire cycle with levels elevating directly after ovulation and staying elevated until menstruation begins. If progesterone levels do not elevate enough after ovulation or drop too soon before menstruation, this can cause a short luteal phase. Here are some of the most common culprits to low progesterone:

Poor Follicle Production
If the pituitary gland does not make enough FSH hormone during the first half of the menstrual cycle (the time between your last period and ovulation), then follicle production may be weak, which can cause a thin uterine lining and an early period. This of course will prohibit fertilization and implantation from taking place.

A Premature Drop in Progesterone
If progesterone levels drop too soon (usually within a few days of ovulation), then the body will automatically think that it is time to flush out the uterus and start all over again. Very short cycles (usually less than 24 days) is usually a sign of this type of luteal phase defect.

Low Lutenizing Hormone
Lutenizing Hormone (LH) increases prior to ovulation occurring (24hrs). It is this spike in LH which causes ovulation to occur. A lower than normal LH surge at ovulation can prevent ovulation from occurring and cause low progesterone levels as well.

Uterine Lining Failure
A fertilized egg needs a nourishing environment to grow into a fetus. This is the job of the uterus. But, if your uterine lining is not thick enough – or strong enough – it can’t sustain this new life and a miscarriage may occur. This to can be caused by low hormones or hormonal imbalance. Estrogen is the hormone which thickens the uterine lining in preparation for implantation and progesterone “ripens” the uterus preparing for implantation.

Abnormally Low Cholesterol Levels & Being Underweight
Another cause of LPD is abnormally low cholesterol levels which results in low to no progesterone production. All hormones, including progesterone, must have cholesterol in order to be manufactured by the body. Being underweight can also be a cause for LPD due to low cholesterol and body fat levels which can cause low hormonal levels across the board.

Solutions for Luteal Phase Defect

While a luteal phase defect can be very serious, prohibiting a pregnancy until it is fixed, the good news is that in most cases it can be helped by natural therapies.

Luteal Phase Defect and low progesterone levels can be increased with a variety of methods including herbs, diet, supplements and/or progesterone cream.

Diet

Make sure that any of the herbal and supplement suggestions mentioned below are made in conjunction with a diet rich in whole foods, specifically:

Vitamin C: A study in Fertility and Sterility showed that vitamin C improves hormone levels and increases fertility in some women with luteal phase defect. During the study, 25% of the women who received vitamin C had got pregnant within 6 months compared to the placebo group in which 11% got pregnant in the same time period. Foods rich in vitamin C are: Papaya, bell peppers, broccoli, brussel sprouts, strawberry and oranges.

Essential fatty acids: EFA’s are important for hormone production. Many women are low in EFA’s, specifically omega 3. Some foods rich in EFA’s are Flaxseeds, walnuts, salmon, sardines, halibut, shrimp, snapper, scallops and chia seeds.

Green leafy vegetables: Green vegetables are rich in B vitamins which are necessary for proper hormonal balance.

Cholesterol from eggs, coconut oil and fat from organic and grass-fed animal products: Cholesterol is necessary for hormone production. Avoid eating a ‘low-fat’ diet and makes sure to eat a diet that includes whole fat sourced from grass-fed animal products. Foods rich in clean cholesterol: grass-fed beef, raw milk from grass-fed cows or goats, whole milk yogurt and kefir, free-range/pastured eggs, butter from grass-fed milk (Kerry Gold is a common one), Coconut oil.

All of these foods are necessary for proper hormone production in the body. Click here to learn more about eating a natural fertility diet…

Vitex (Chasteberry)

For those looking for a more natural way to boost their progesterone levels, taking a supplement of Vitex (otherwise known as Chasteberry) may be the answer. A small fruit tree grown in the Mediterranean, Vitex has been used for centuries to treat all sorts of female issues including infertility. Studies have shown this herb to be affective at lengthening the luteal phase.

While it does not contain any hormones itself, it does help the body to increase its own production of luteinizing hormone (promoting ovulation to occur) which in turns boosts progesterone levels during the luteal phase of the cycle.

Progesterone Cream

One of the most common treatments for lengthening a woman’s luteal phase is to use a progesterone cream. Found over the counter, natural progesterone cream should be used twice a day on the inner arm, inner thigh, or neck, after ovulation has occurred until the period begins. If the problem begins with low Lutenizing Hormone, then just adding progesterone may not work. Click here to learn more about using progesterone cream properly…

B6

A lot of women have reported a lengthened luteal phase after supplementing with B6. This may have been caused by the hormonal balancing effect B6 has on the body. B6 can be found in tuna, bananas, turkey, liver, salmon and many of the greens. Taking a B6 supplement is also another choice. Suggested usage is 50mg up to 100mg daily. Make sure to use B6 in conjunction with a B complex or multivitamin to avoid causing imbalances.

Antioxidants

Recent studies have found that oxidative damage may be a cause of luteal phase defect. Women who had luteal phase defect and recurrent miscarriages were found to have significantly lower levels of antioxidants than healthy women. There are so many benefits of antioxidants on fertility, they should be a part of every couples fertility program. Learn more about antioxidants and fertility…

Any type of luteal Phase Defect is serious since it will prevent pregnancy. Luckily, they can be easily remedied with the therapies listed above. You may also find it helpful to get your hormones tested as well as fertility chart to determine the length of your luteal phase and to also see the changes your chosen protocol are causing.

References:
1. Weiss, R.F. 1988. Herbal Medicine, Beaconsfield Arcanum, Gothenburg, Sweden.
2. Trickey, R. Women’s Hormones & the Menstrual Cycle. 2003, Allen & Unwin.
3. Vitamin C Increases Fertility in Women with Luteal Phase Defect. Fertility and Sterility (2003;80:459–61)